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Rabbit Anti-COX10 Antibody (CSB-PA621520ESR1HU)
Supplier: CUSABIO BIOTECH CO.
Protoheme IX farnesyltransferase Antibody,mitochondrial Antibody,Heme O synthase Antibody,COX10 AntibodyMore alternative names for the antibody
2410004F01Rik antibody|AU042636 antibody|COX10 antibody|COX10_HUMAN antibody|Cytochrome c oxidase assembly protein antibody|Cytochrome c oxidase subunit X antibody|Heme A farnesyltransferase antibody|Heme O synthase antibody|OTTMUSP00000006085 antibody|Protoheme IX farnesyltransferase, mitochondrial antibody|Protoheme IX farnesyltransferase, mitochondrial precursor antibody|RP23-78H18.1 antibody
Anti-COX10 antibody (ab84053)
Anti-COX10 antibody (ab84053)
Recommended applications: ELISA, WB, IHC
Recommended dilution: User optimized
Recommended protocols: check protocols
|Catalogue No.|| |
Recombinant human Protoheme IX farnesyltransferase, mitochondrial protein (1-160AA)
ELISA, WB, IHC
|Recommended dilution|| |
|Molecular weight|| |
Antigen Affinity Purified
Shipped at 4 Celcius Degree. Upon delivery aliquot and store at -20 Celcius Degree or -80 Celcius Degree. Avoid repeated freeze.
|Database links|| |
Entrez Gene:417329( Chicken), Entrez Gene:511440( Cow), Entrez Gene:100073118( Horse), Entrez Gene:1352( Human), Entrez Gene:70383( Mouse), Entrez Gene:363617( Rat), Entrez Gene:553384 ( Zebrafish), Omim:602125( Human), SwissProt:Q12887( Human), SwissProt:Q8CFY5( Mouse), Unigene:462278( Human), Unigene:340211( Mouse)
|Protein function|| |
Converts protoheme IX and farnesyl diphosphate to heme O. .
|Involvement in disease|| |
Mitochondrial complex IV deficiency (MT-C4D) [MIM:220110]: A disorder of the mitochondrial respiratory chain with heterogeneous clinical manifestations, ranging from isolated myopathy to severe multisystem disease affecting several tissues and organs. Features include hypertrophic cardiomyopathy, hepatomegaly and liver dysfunction, hypotonia, muscle weakness, exercise intolerance, developmental delay, delayed motor development and mental retardation. Some affected individuals manifest a fatal hypertrophic cardiomyopathy resulting in neonatal death. A subset of patients manifest Leigh syndrome. . Note=The disease is caused by mutations affecting the gene represented in this entry.; Leigh syndrome (LS) [MIM:256000]: An early-onset progressive neurodegenerative disorder characterized by the presence of focal, bilateral lesions in one or more areas of the central nervous system including the brainstem, thalamus, basal ganglia, cerebellum and spinal cord. Clinical features depend on which areas of the central nervous system are involved and include subacute onset of psychomotor retardation, hypotonia, ataxia, weakness, vision loss, eye movement abnormalities, seizures, and dysphagia. Note=The disease is caused by mutations affecting the gene represented in this entry.
|Protein sequence and domain|| |
Belongs to the UbiA prenyltransferase family.
|Protein cellular localization|| |
Mitochondrion membrane; Multi-pass membrane protein.
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CUSABIO BIOTECH CO.
|Product type|| |
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