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Mouse Monoclonal GFAP antibody [6A6] (STJ98106)
Supplier: St John’s Laboratory Ltd.
Recommended applications: WB, IHC, IF, ELISA
Recommended dilution: WB 1:500-1:2000; IHC 1:200-1:1000; IF 1:200-1:1000; ELISA 1:10000
Recommended protocols: check protocols
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Check alternative names for the antibodyExpand
GFAP antibody,|wu:fb34h11 antibody|ALXDRD antibody|cb345 antibody|etID36982.3 antibody|FLJ42474 antibody|FLJ45472 antibody|GFAP antibody|GFAP_HUMAN antibody|gfapl antibody|Glial fibrillary acidic protein antibody|Intermediate filament protein antibody|wu:fk42c12 antibody|xx:af506734 antibody|zgc:110485 antibody|Anti-GFAP antibody (ab53554)
SCBT cat No: sc-32955|
GFAP Monoclonal Antibody
|Catalogue No.|| |
GFAP Monoclonal Antibody detects endogenous levels of GFAP protein.
Purified recombinant fragment of human GFAP expressed in E Coli
WB, IHC, IF, ELISA
|Recommended dilution|| |
WB 1:500-1:2000; IHC 1:200-1:1000; IF 1:200-1:1000; ELISA 1:10000
GFAP Antibody was tube-contained. Ascitic fluid containing 0.03% sodium azide.
GFAP Antibody was purified using affinity purification.
-20 Celsius degree. Avoid repeated freeze/thaw cycles.
|Alternative antibody names|| |
Glial fibrillary acidic protein antibody, GFAP antibody
|Protein names|| |
Glial fibrillary acidic protein , GFAP
|Protein function|| |
GFAP, a class-III intermediate filament, is a cell-specific marker that, during the development of the central nervous system, distinguishes astrocytes from other glial cells.
|Protein tissue specificity|| |
Expressed in cells lacking fibronectin.
|Involvement in disease|| |
Alexander disease (ALXDRD) [MIM:203450]: A rare disorder of the central nervous system. The most common form affects infants and young children, and is characterized by progressive failure of central myelination, usually leading to death within the first decade. Infants with Alexander disease develop a leukodystrophy with macrocephaly, seizures, and psychomotor retardation. Patients with juvenile or adult forms typically experience ataxia, bulbar signs and spasticity, and a more slowly progressive course. Histologically, Alexander disease is characterized by Rosenthal fibers, homogeneous eosinophilic inclusions in astrocytes. . Note: The disease is caused by mutations affecting the gene represented in this entry.
|Protein sequence and domain|| |
Belongs to the intermediate filament family.
|Protein post-translational modifications|| |
Phosphorylated by PKN1.
|Protein cellular localization|| |
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St John’s Laboratory Ltd.
|Product type|| |
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