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Rabbit Polyclonal Phospho-hnRNP K (S216) antibody (STJ90631)
Supplier: St John’s Laboratory Ltd.
Recommended applications: WB, ELISA
Recommended dilution: WB 1:500-1:2000; ELISA 1:20000;
Recommended protocols: check protocols
Click or hover above images to see image description for hnRNP K (phospho Ser216) Polyclonal Antibody.
Check alternative names for the antibodyExpand
HNRNPK antibody, HNRPK antibody,|CSBP antibody|dC stretch binding protein antibody|FLJ41122 antibody|Heterogeneous nuclear ribonucleoprotein K antibody|hnRNP K antibody|HNRNPK antibody|HNRPK antibody|HNRPK_HUMAN antibody|Transformation up regulated nuclear protein antibody|Transformation up-regulated nuclear protein antibody|Transformation upregulated nuclear protein antibody|TUNP antibody|Anti-hnRNP K antibody [3C2] – ChIP Grade (ab39975)
SCBT cat No: sc-28380|sc-53620|sc-25373|sc-16554|sc-32307|sc-32317|
hnRNP K (phospho Ser216) Polyclonal Antibody
|Catalogue No.|| |
Human, Mouse, Rat
Phospho-hnRNP K (S216) Polyclonal Antibody detects endogenous levels of hnRNP K protein only when phosphorylated at S216.
Synthesized phospho-peptide derived from hnRNP K (phospho Ser216) at AA range 160-240
|Recommended dilution|| |
WB 1:500-1:2000; ELISA 1:20000;
|Molecular weight|| |
hnRNP K (phospho Ser216) Antibody was tube-contained. Liquid in PBS containing 50% glycerol, 0.5% BSA and 0.02% sodium azide.
hnRNP K (phospho Ser216) Antibody was affinity-purified from rabbit antiserum by affinity-chromatography using epitope-specific immunogen.
-20 Celsius degree. Avoid repeated freeze/thaw cycles.
|Alternative antibody names|| |
Heterogeneous nuclear ribonucleoprotein K antibody, hnRNP K antibody, Transformation up-regulated nuclear protein antibody, TUNP antibody
|Protein names|| |
Heterogeneous nuclear ribonucleoprotein K , hnRNP K , Transformation up-regulated nuclear protein , TUNP
|Involvement in disease|| |
Au-Kline syndrome (AUKS) [MIM:616580]: A disorder characterized by intellectual disability, facial dysmorphism, cardiac defects, and connective tissue and skeletal abnormalities. Dysmorphic features include long palpebral fissures, ptosis, a broad prominent nasal bridge, hypoplastic alae nasi, an open downturned mouth, ears with underdeveloped and thick helices, high palate, and a unique tongue with a prominent median crease. Hypotonia, hyporeflexia, and high pain tolerance are additional features. . Note: The disease is caused by mutations affecting the gene represented in this entry.
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St John’s Laboratory Ltd.
|Product type|| |
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