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Rabbit Polyclonal Phospho-p63 (S395) antibody (STJ90646)
Supplier: St John’s Laboratory Ltd.
Recommended applications: WB, ELISA
Recommended dilution: WB 1:500-1:2000; ELISA 1:40000;
Recommended protocols: check protocols
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Check alternative names for the antibodyExpand
TP63 antibody, KET antibody, P63 antibody, P73H antibody, P73L antibody, TP73L antibody,|AIS antibody|ANDR_HUMAN antibody|Androgen nuclear receptor variant 2 antibody|Androgen receptor (dihydrotestosterone receptor; testicular feminization; spinal and bulbar muscular atrophy; Kennedy disease) antibody|Androgen receptor antibody|androgen receptor splice variant 4b antibody|AR antibody|AR8 antibody|DHTR antibody|Dihydro testosterone receptor antibody|Dihydrotestosterone receptor (DHTR) antibody|Dihydrotestosterone receptor antibody|HUMARA antibody|HYSP1 antibody|KD antibody|Kennedy disease (KD) antibody|NR3C4 antibody|Nuclear receptor subfamily 3 group C member 4 (NR3C4) antibody|Nuclear receptor subfamily 3 group C member 4 antibody|SBMA antibody|SMAX1 antibody|Spinal and bulbar muscular atrophy (SBMA) antibody|Spinal and bulbar muscular atrophy antibody|Testicular Feminization (TFM) antibody|TFM antibody|Anti-p63 antibody [EPR5701] (ab124762)
SCBT cat No: To be updated
p63 (phospho Ser395) Polyclonal Antibody
|Catalogue No.|| |
Human, Mouse, Rat
Phospho-p63 (S395) Polyclonal Antibody detects endogenous levels of p63 protein only when phosphorylated at S395.
Synthesized phospho-peptide derived from p63 (phospho Ser395) at AA range 330-410
|Recommended dilution|| |
WB 1:500-1:2000; ELISA 1:40000;
|Molecular weight|| |
p63 (phospho Ser395) Antibody was tube-contained. Liquid in PBS containing 50% glycerol, 0.5% BSA and 0.02% sodium azide.
p63 (phospho Ser395) Antibody was affinity-purified from rabbit antiserum by affinity-chromatography using epitope-specific immunogen.
-20 Celsius degree. Avoid repeated freeze/thaw cycles.
|Alternative antibody names|| |
Tumor protein 63 antibody, p63 antibody, Chronic ulcerative stomatitis protein antibody, CUSP antibody, Keratinocyte transcription factor KET antibody, Transformation-related protein 63 antibody, TP63 antibody, Tumor protein p73-like antibody, p73L antibody, p40 antibody, p51 antibody
|Protein names|| |
Tumor protein 63 , p63 , Chronic ulcerative stomatitis protein , CUSP , Keratinocyte transcription factor KET , Transformation-related protein 63 , TP63 , Tumor protein p73-like , p73L , p40 , p51
|Involvement in disease|| |
Acro-dermato-ungual-lacrimal-tooth syndrome (ADULT syndrome) [MIM:103285]: A form of ectodermal dysplasia. Ectodermal dysplasia defines a heterogeneous group of disorders due to abnormal development of two or more ectodermal structures. ADULT syndrome involves ectrodactyly, syndactyly, finger- and toenail dysplasia, hypoplastic breasts and nipples, intensive freckling, lacrimal duct atresia, frontal alopecia, primary hypodontia and loss of permanent teeth. ADULT syndrome differs significantly from EEC3 syndrome by the absence of facial clefting. . Note: The disease is caused by mutations affecting the gene represented in this entry.; Ankyloblepharon-ectodermal defects-cleft lip/palate (AEC) [MIM:106260]: An autosomal dominant condition characterized by congenital ectodermal dysplasia with coarse, wiry, sparse hair, dystrophic nails, slight hypohidrosis, scalp infections, ankyloblepharon filiform adnatum, maxillary hypoplasia, hypodontia and cleft lip/palate. . Note: The disease is caused by mutations affecting the gene represented in this entry.; Ectrodactyly, ectodermal dysplasia, and cleft lip/palate syndrome 3 (EEC3) [MIM:604292]: A form of ectodermal dysplasia, a heterogeneous group of disorders due to abnormal development of two or more ectodermal structures. It is an autosomal dominant syndrome characterized by ectrodactyly of hands and feet, ectodermal dysplasia and facial clefting. . Note: The disease is caused by mutations affecting the gene represented in this entry.; Split-hand/foot malformation 4 (SHFM4) [MIM:605289]: A limb malformation involving the central rays of the autopod and presenting with syndactyly, median clefts of the hands and feet, and aplasia and/or hypoplasia of the phalanges, metacarpals, and metatarsals. Some patients have been found to have mental retardation, ectodermal and craniofacial findings, and orofacial clefting. . Note: The disease is caused by mutations affecting the gene represented in this entry.; Limb-mammary syndrome (LMS) [MIM:603543]: Characterized by ectrodactyly, cleft palate and mammary-gland abnormalities. . Note: The disease is caused by mutations affecting the gene represented in this entry.; Note: Defects in TP63 are a cause of cervical, colon, head and neck, lung and ovarian cancers.; Ectodermal dysplasia, Rapp-Hodgkin type (EDRH) [MIM:129400]: A form of ectodermal dysplasia, a heterogeneous group of disorders due to abnormal development of two or more ectodermal structures. Characterized by the combination of anhidrotic ectodermal dysplasia, cleft lip, and cleft palate. The clinical syndrome is comprised of a characteristic facies (narrow nose and small mouth), wiry, slow-growing, and uncombable hair, sparse eyelashes and eyebrows, obstructed lacrimal puncta/epiphora, bilateral stenosis of external auditory canals, microsomia, hypodontia, cone-shaped incisors, enamel hypoplasia, dystrophic nails, and cleft lip/cleft palate. . Note: The disease is caused by mutations affecting the gene represented in this entry.; Non-syndromic orofacial cleft 8 (OFC8) [MIM:129400]: A birth defect consisting of cleft lips with or without cleft palate. Cleft lips are associated with cleft palate in two-third of cases. A cleft lip can occur on one or both sides and range in severity from a simple notch in the upper lip to a complete opening in the lip extending into the floor of the nostril and involving the upper gum. Note: The disease is caused by mutations affecting the gene represented in this entry.
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St John’s Laboratory Ltd.
|Product type|| |
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